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Strong Effect Analgesic & Antipyretic 4-Acetamidophenol ( Paracetamol ) Powder

Categories Pharmaceutical Raw Materials
Brand Name: TINGYI
Model Number: CAS: 103-90-2
Certification: GMP, SGS , ISO 9001:2008 , KOSHER
Place of Origin: China
MOQ: 100g
Price: Negotiable ( Discounts For Big Order )
Payment Terms: Western Union, MoneyGram, Bank Transfer, Bitcoin
Supply Ability: 3000 KG/Month
Delivery Time: Within 7 Working Days
Packaging Details: Stealth And Discreet Packaging
Product Name: 4-Acetamidophenol
Other Name: Paracetamol
MF: C8H9NO2
MW: 151.16
EINECS: 203-157-5
MP: 168-172 °C
FP: 11 °C
Density: 1.293 g/cm3
Water Solubility: 14 g/L (20°C)
Appearance: white crystalline solid
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    Strong Effect Analgesic & Antipyretic 4-Acetamidophenol ( Paracetamol ) Powder


    Strong Effect Analgesic & Antipyretic 4-Acetamidophenol (Paracetamol) Powder



    Description:


    This product is acetanilide antipyretic analgesics. By inhibiting ring oxidase and


    selectivity in the hypothalamus of the temperature regulating center, the synthesis of


    prostaglandins in peripheral vascular expansion, sweating and antipyretic effect, strength of


    its antipyretic effect similar to aspirin.

    Paracetamol is a widely used over-the-counter analgesic (pain reliever) and antipyretic


    (fever reducer). Acetaminophen is the name adopted for this pharmacologic agent in the U. S.


    (USAN) and Japan; Paracetamol is approved in a variety of international venues.

    Paracetamol is classified as a mild analgesic. It is commonly used for the relief of


    headaches and other minor aches and pains and is a major ingredient in numerous cold and flu


    remedies. In combination with opioid analgesics, paracetamol can also be used in the


    management of more severe pain such as post-surgical pain and providing palliative care in


    advanced cancer patients. Though paracetamol is used to treat inflammatory pain, it is not


    generally classified as an NSAID because it exhibits only weak anti-inflammatory activity.



    Application:

    Paracetamol is approved for reducing fever in people of all ages.

    Paracetamol is used for the relief of pains associated with many parts of the body. It has


    analgesic properties comparable to those of aspirin, while its antiinflammatory effects are


    weaker. It is better tolerated than aspirin in patients in whom excessive gastric acid


    secretion or prolongation of bleeding time may be a concern.

    Paracetamol powder nutritional supplemen Mainly used for amino acid infusion,due to special


    wettability, also used in cold cream,osmetics. Serine constitute the human body of several


    kinds of essential amino acid of protein a, for the construction of protein has a very


    important role.


    Research Result:


    Since their synthesis in the late 1800s paracetamol (acetaminophen) and phenacetin have


    followed divergent pathways with regard to their popularity as mild analgesic/antipyretic drugs.


    Initially, paracetamol was discarded in favour of phenacetin because the latter drug was


    supposedly less toxic. Today the opposite is true, and paracetamol, along with aspirin, has


    become one of the two most popular 'over-the-counter' non-narcotic analgesic agents. This


    marked increase in the wide approval attained by paracetamol has been accompanied by the


    virtual commercial demise of phenacetin because of its role, albeit somewhat circumstantial, in


    causing analgesic nephropathy. Both paracetamol and phenacetin are effective mild analgesics,


    suitable for treating mild to moderate pain, and their actions are broadly comparable with those


    of aspirin and related salicylates, although they do not appear to possess significant anti-


    inflammatory activity. Since a major portion of a dose of phenacetin is rapidly metabolised to


    paracetamol, it seems possible that phenacetin owes some of its therapeutic activity to its main


    metabolite, paracetamol, whereas its most troublesome side effect (methaemoglobinaemia) is


    due to another metabolite, p-phenetidine. The mechanism of action of paracetamol is poorly


    defined, although it has been speculated that it may selectively inhibit prostaglandin production in


    the central nervous system, which would account for its analgesic/antipyretic properties. The lack


    of any significant influence on peripheral cyclooxygenase would explain the absence of anti-


    inflammatory activity. At therapeutic doses paracetamol is well tolerated and produces fewer side


    effects than aspirin. The most frequently reported adverse effect associated with paracetamol is


    hepatotoxicity, which occurs after acute overdosage (usually doses greater than 10 to 15g are


    needed) and, very rarely, during long term treatment with doses at the higher levels of the


    therapeutic range. Paracetamol damages the liver through the formation of a highly reactive


    metabolite which is normally inactivated by conjugation with glutathione. Overdoses of


    paracetamol exhaust glutathione stores, thus allowing the accumulation of this toxic metabolite


    which covalently binds with vital cell elements and can result in liver necrosis. Glutathione


    precursors (notably intravenous N-acetylcysteine) have proved remarkably successful in treating


    paracetamol overdose, as long as treatment is initiated within 10 hours.




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